Nutrition and Disease

 

Unfortunately, cases of nutritional disease are increasing throughout the country. For this discussion, you are asked to share an example of someone you know who has a nutritional disease.

Include the following in your reflection:

Describe someone you know, whether a family member, friend, coworker or peer, that has a nutrition-related disease.
Provide some basic characteristics such as age, identified gender, living status (alone or with someone).
What is the disease/disorder?
How is the disease related to nutrition? Support your response with an external reference.
What are the eating habits of the person described?
How might the eating habits have contributed to the disease?
If this person was your patient, what would your recommendations be?
What eating habits would you recommend be changed?
List a video, blog, or website you might suggest your patient access for help making this change

Sample Solution

Nutritional disease, any of the nutrient-related diseases and conditions that cause illness in humans. They may include deficiencies or excesses in the diet, obesity and eating disorders, and chronic diseases such as cardiovascular disease, hypertension, cancer, and diabetes mellitus. Nutritional diseases also include developmental abnormalities that can be prevented by diet, hereditary metabolic disorders that respond to dietary treatment, the interaction of foods and nutrients with drugs, and potential hazards in the food supply. Nutritional diseases include, but are not limited to, protein energy malnutrition, scurvy, rickets, beriberi, and vitamin K deficiency. Vitamin K deficiency can contribute to significant bleeding, poor bone development, osteoporosis, and increased risk of cardiovascular disease.

or on the other hand fostering a coagulation. He additionally referenced his broad medical clinic and careful history. He said that he has had 32 techniques which might have come about in harmed vasculature. This injury to the vasculature is a region of the vessel that could shape a clots that could burst. The different medical procedures and hospitalizations for the repetitive UTIs likewise increment his stability as talked about before. He is likewise decently stout which is a component that expands his gamble for blood clot development too. Injury is likewise a significant gamble factor for fostering a PE and he was engaged with an engine vehicle mishap which brought about his spinal injury. He didn’t indicate on the off chance that he had any familial blood condition which would influence thickening. A more definite family ancestry would be important to evaluate his gamble of a coagulating issue.

Clinical Discoveries in the Set of experiences and Physical of Aspiratory Embolism:

While assessing a patient for a PE, it is famously troublesome on the grounds that the normal signs and side effects are profoundly factor and vague for PE. The most widely recognized side effect in patients giving PE is dyspnea with pleuritic chest torment (uptodate). The Imminent Examination of PE Analysis study took a gander at normal giving signs and side effects in patients intense PE. They included dyspnea (73%), inspiratory chest torment (66%), hack (37%), leg torment (26%), hemoptysis (13%), palpitations (10%), wheezing (9%), angina torment (4%), respiratory rate >20 (70%), snaps (51%), pulse >100 (30%), fourth heart sound (S4) (24%), highlighted P2 heart sound (23%), temperature >38.5C (7%), Homans sign (4%), pleural grating rub (3%), third heart sound (3%), cyanosis (1%) (lange). In the review, 97% of patients had no less than one of these three discoveries: dyspnea, chest torment with breathing, or tachypnea (lange). Nonetheless, as found in the wide assortment of signs and side effects, the clinical picture that the patient presents with could be tremendously divergent. Along these lines, clinical choice devices have been formed to utilize data that the patient can give to make a more instructed evaluation of the probability that the patient is having an intense PE. A portion of these instruments are examined beneath. Shock and blood vessel hypotension are two clinical discoveries that are uncommon however critical to distinguish as they demonstrate a focal PE and an absence of hemodynamic save (3).

An electrocardiogram (ECG) is a device that can be utilized in the work up of a patient who is encountering intense chest torment that might result from a PE. While there are no signs that are analytic of intense PE, it is valuable in precluding different reasons for chest agony like myocardial localized necrosis or pericarditis. Having said that, 70% of ECG brings about patients encountering intense PEs are strange yet most are vague (lange). The most widely recognized irregularity seen is sinus tachycardia (lange) yet one more typical finding is T-wave reversal in drives V1-V4 which is generally ordinarily connected with the seriousness of the PE (13). Other exemplary discoveries related with PE incorporate S1Q3T3 (S wave in lead I, Q wave in lead III, and reversed T wave in lead III) and right pack branch block both demonstrating right ventricular strain.

Chest x-beam is one more test that is regularly finished to preclude different reasons for the introducing side effects. There isn’t anything that is indicative of PE on chest x-beam, yet there are a few signs that are reminiscent of the finding. The three signs are Westermark sign, Fleishner sign, and Hampton bump. The Westermark sign is a sharp cut off in the pneumonic vasculature that outcomes from enlargement of the aspiratory supply route proximal to the embolism and a breakdown of the vasculature distal to the embolism. Fleishner sign (additionally called the knuckle sign) is an expansion in the pneumonic conduit proximal to the embolism. The Hampton bump is a wedge-molded darkness in the lung that is optional to infracting tissue because of a PE. While these signs are normal for intense PE, they are not regularly seen.

HM was breathing easily and had typical breath sounds without any snaps or wheezes and had an ordinary respiratory rate. He has no chest torment or hack which are normal introducing side effects of PE.

Work up:

The supplier should have a high file of doubt of PE and answer accordingl

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