Option I:
You have been hired by President Trump to control the coronavirus pandemic. He has given you broad authority to implement any policies/programs that you think would minimize the spread of the disease and, of course, reduce the number of Americans dying from the virus to the absolute minimum.
write a 5-page paper, using some of the concepts we learned in this class, detailing exactly how you would accomplish reducing the number of people who not only contract Covid-19, but for those who do, how you would reduce the severity of disease and lower death rates.
Some things you might want to mention:
– what is the coronavirus?
– what are typical symptoms?
– who is most at risk of becoming seriously ill or dying?
– how can the spread of coronavirus be minimized?
– would you focus your resources the same across the nation or would you focus on certain areas of the country?
– what agencies (federal, state, local) would you include in your response efforts?
– would you try anything new? Think outside the box?
– etc., etc., etc.
Option II:
Select any Environmental Health topic of your choice and write a 5-Page paper. For example, you decide to write on Foodborne Illness Outbreaks on cruise ships and decide to discuss: 1) Common foodborne illness pathogens encountered on cruise ships, 2) How can these illnesses be prevented, 3) A few examples of recent, documented outbreaks (e.g., within the last 5 years), 4) Common foods and/or ways foodborne illness is transmitted on cruise ships, and recommendations on implementation measures that the cruise industry can take to significantly reduce foodborne illness (and maybe disease transmission in general) incidence rates. I am using foodborne illness as just one example of an environmental health topic you can write on – feel free to choose any environmental health topic that you find interesting or want to write about.
Please cite your sources. Usually I require at least 5 peer-reviewed journal articles but if you choose to write on a unique topic and can’t find 5 sources please contact me so we can discuss your options. I don’t care what system you use to cite your sources (Turabian, MLA, APA, etc.) as long as I can access the source and verify your reference.
Also, refer to the syllabus for information on formatting/stylistic questions. I will post an Announcement about this assignment so please direct any and all questions about the assignment to this week’s announcement so every student will have access to the information.
polymorphism had impaired cognitive abilities (Bennett et al., 2016). This polymorphism has been linked to a decrease in hippocampus volume and prefrontal cortex gray matter (Bagnato et al., 2012). Similar results have been revealed in patients following brain injury. This suggests that patients with this polymorphism would be more susceptible to poor outcomes and disorders of consciousness. However, Bagnato et al revealed that the Val66Met polymorphism may not affect the improvement of cognitive abilities in patients in a vegetative state following traumatic brain injuries. The percentage of patients with and without the Val66Met polymorphism who emerged from the vegetative state did not statistically differ. Additionally, the cognitive functions were similar between these patients (Bagnato et al., 2012). Therefore, although polymorphisms in BDNF impact brain structures and are implicated in impaired cognition, it does not seem to have a prominent effect on the recovery or cognition of patients in a vegetative state.
The effect of these polymorphisms on the outcomes following brain injuries imply that there is a genetic susceptibility factor that can make an individual more at risk of entering a vegetative and minimally conscious state
There are few studies that have elucidated the role of subcellular organelles in vegetative and minimally conscious states. However, diseases, such as Alzheimer’s disease, that have organelle dysfunction sometimes progress into vegetative and minimally conscious states. Alzheimer’s disease is a common type of cortical dementia that is characterized by severe memory impairments and mostly affects people over the age of 65. Mitochondrial dysfunction plays a key role in the onset of this disease (Moreira et al., 2010). In healthy individuals, mitochondria are involved in proper neuron function by generating adenosine triphosphate (ATP) for energy needs and monitoring cellular health. Dysfunctions in mitochondria result in a decreased production of ATP and a released of pro-apoptotic factors (Moreira et al., 2010). Additionally, impaired mitochondria are unable to serve as a intracellular calcium buffer, which is required for calcium homeostasis and neuron function. As a result, cell death and neurodegeneration can occur, which contributes to the loss of cognitive abilities seen in Alzheimer’s patients (Moreira et al., 2010). Due to neurodegeneration, 4-12 years after a patient has been diagnosed with Alzheimer’s, they can progress into a vegetative state (Heston et al., 1977). A study conducted by Jaul et al. revealed that 30.5% of elderly patients with dementia had persistent vegetative state (Jaul et al., 2007). However, a study done by Volicer et al demonstrated that it is unlikely for Alzheimer’s disease to progress into a vegetative state (Volicer et al., 1997). Both studies noted the difficulty in diagnosing patients with Alzheimer’s disease, since the cognitive abilities are not stable and can differ from patient to patient. Additionally, it was suggested that th