Biventricular heart failure occurs when both ventricle function of the heart is impaired. The manifestations is a combination of right-sided and left-sided heart failure.
Diastolic dysfunction of the heart is also referred to as heart failure with preserved LVEF. According to Figueroa & Peters (2006), the diastolic function is determined a passive process, elasticity of the left ventricle and an active process, the process of myocardial relaxation. The left ventricular filling generates a suction effect when relaxing the myocardium. Ventricular filling is impaired due to ”..restrictive, obstructive, non-obstructive, hypertrophic, and infiltrative cardiomyopathies(Osborn, 2014 p1028).” It is diagnosed when patients have a normal LVEF but manifest the symptoms of heart failure.
(Osborn, 2014 p1028-1030)
MI
Myocardial infarction results from necrosis of myocardial tissue caused by relative or absolute lack of blood supply to the myocardium. Apoptosis results in a permanent loss of myocardial muscle function. Most acute MI are caused by atherosclerosis, which results in plaque formation within the coronary arteries. Plaque deposition that results in endolelial changes causes narrowing of the lumen of the coronoary artery. If an unstable plaque ruptures, the immune system responsds with localized infection; platelets aggregate at the site of injured plaque and a thrombus forms. If the lesion is large enough to fill the vessel lumen, then it will result in a occlusion of blood flow. Occlusion can also be caused by coronary artery vasospasm.
Non-ST segment elevation myocardial infarction(NSTEMI) shares a common pathophysiology and clinical presentation as unstable angina because both usually occur as a result of transient subtotal occlusion of a coronary artery with reduced coronary blood flow resulting from plaque disruption. It can occur when perfusion pressure is low, and in sudden hypotension, or when there is increased myocardial workload, as in aortic st