tiation of PR programme such as the fraction of oxygen saturated haemoglobin in the arteries relative to the total haemoglobin by arterial blood gas tests and tracking dyspnea usuing the Borg CR10 Scale that measures physical activity intensity levels (Borg, 1982).

In some cases where patients have lost a lot of lung function due to severe COPD modified exercises are established to cater for individual patients with varying levels of lung function.

Aeroblic Exercise

Skeletal muscle dysfunction is influenced by skeletal muscle strength and endurance function and structure (fiber size, fiber type distribution, capillary density, and metabolic capacity) (Zeng et al., 2018). A prolonged lack of exercise promotes a decrease in physical activity in COPD sufferers. The activity of these muscles rely greatly on the physiologic structural elements of the particular muscle and the velocity at which a fiber contracts inversely regulates the fibers ability t resist fatigue.

Type I fibers are slow-twitch fibers and can endure long periods of exercise as they are highly resistant to fatigue. They are comprised of myosin heavy-chain (MyHC) type I. Type IIx fibers are fast-twitch fibers and are a subset or the fast twitch muscles which contain type IIa and type IIb. Type IIx fibers are the strongest and lowest endurance abibity as they fatigue much easier. Their composition differs from Type I fibers as they are composed of MyHC type IIx. It has been reported that a less than 27% of fiber type I proportionality is regarded as unusually low. Additionally, an amount of fiber type IIX that is greater than 29% is regarded as unusually large (Barreiro and Gea, 2016). In cases where the subject suffers from COPD, the amount of type I fibers are significantly lower than that of a hea