The courts have established a distinction between “testimonial” and “non-testimonial” statements

 

 

The courts have established a distinction between “testimonial” and “non-testimonial” statements determining whether an objective person would be aware or expect to use those statements later in a trial to be considered “testimonial” (Cotchett, 2022). To determine statement’s admissibility as evidence at trial the Court’s rationale in Davis v. Washington holds that statements are considered “testimonial” when they are made with the primary purpose of creating an out-of-court substitute for trial testimony (2006). The key consideration is whether the statement is made with the expectation that it will be used in a criminal prosecution.

In this case, the woman’s text messages, and photograph were sent to her friend with the intention of seeking assistance and documenting the assault by her ex-husband. This would likely be considered testimonial in nature. The exchange of messages about the identity of the assailant and the woman’s location can be seen as a substitute for trial testimony, aimed at providing evidence for a potential criminal prosecution. If the statements are deemed testimonial, the admission of such evidence at trial would require compliance with the Confrontation Clause, which guarantees the right of the accused to confront and cross-examine witnesses against them. Since the woman is unable to appear at trial, the defendant may argue that his right to confront the witness is violated under the Confrontation Clause of the Sixth Amendment, which stated that “in all criminal prosecutions, the accused shall enjoy the right to be confronted by the witnesses against him” (United States v. Bench, n.d).

After reading Deuteronomy 19:15, my opinion on this case would not change. In a way, I consider the helping friend, the text messages, and the photographs to be extra witnesses to this case. Messages and photographs can leave a technological footprint, such as timestamps that are not modifiable, which can provide very valuable evidence on a case. The friend can also provide testimony in court, being truthful about the events like the Bible mandates on 2 Timothy 2:15 stating “Do your best to present yourself to God as one approved, a worker who does not need to be ashamed and who correctly handles the word of truth” (NIV, 2011). In conclusion, there would be more than one witness to this offense.

 

Sample Solution

NAc receive dopamine (DA) projections from the ventral tegmental area (VTA) (Björklund and Dunnett, 2007, Ikemoto, 2007, Morales and Margolis, 2017) and this pathway play a major role in motivated behaviours, reinforcement learning and reward processing (Hamid et al., 2016; Salamone and Correa, 2012; Schultz, 2016; Watabe-Uchida et al., 2017). Like any other process, there are negative feedback pathways to balance the projections and prevent overexpression of DA. This arise from various structures (Matsui et al., 2014) but recent studies show that NAc is the main source of this inhibitory input (GABAergic input) (Beier et al., 2015; Watabe-Uchida et al., 2012). There were few conflicting results on this with studies suggesting inputs from NAc to VTA to be disinhibiting (Bocklisch et al., 2013; Chuhma et al., 2011; Xia et al., 2011) and a recent study addressing that NAc synapse onto VTA GABA as well as DA neurons via GABA-A receptor (GABAAR) and GABA-B receptor (GABABR) respectively (Edward et al., 2017). This, however, also projects a different result compared to the study done by Paladini in 1999 where inhibitory responses from the striatum to DA neurons were blocked by GABA-A antagonist hinting at pathway mediated by GABA-A instead.

In this particular study by Hongbin et al. in 2017, the shell component of the NAc is further subdivided into medial shell (NAcMed) and lateral shell (NAcLat). D1-MSN in the NAcMed is found inhibiting NAcMed-projecting DA neurons via GABAAR while NAcLat-projecting DA neurons via GABABR. D1-MSNs in the NAcLat, on the other hand, projects onto VTA GABA to result in disinhibition of NAcL

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