Understanding of Karl Marx’s theories of exploitation under capitalism

To consolidate your understanding of Karl Marx’s theories of exploitation under capitalism as well as different approaches to the concepts of commodity fetishism and ideology, watch the required documentary, David Redmon’s Mardi Gras: Made in China (2005), which tracks the production and consumption of Mardi Gras beads. After watching the documentary, draw on Part IV of Unit 6’s unit instructional notes, Part II of Unit 7’s unit instructional notes, and related arguments in the textbook and the additional required reading by Gary Lapon, “What Do We Mean By Exploitation,” to write a brief essay answering the following questions. Note that to answer these questions and succeed on this assignment, you are strongly advised to watch the documentary more than once and take notes as you watch it.

In your own words, explain Marx’s theory of how exploitation works under capitalism. To answer this question, you will need to explain the differences between use-value, exchange-value, and surplus-value, what produces value according to Marx, and how capitalism reproduces exploitation and inequality. Next, describe the conditions of labour in which Chinese workers are producing the Mardi Gras beads, as well as the roles in and justifications for this labour given by the Chinese factory owner, Roger Wong, and the American distributor, Dom Carlone. How would Marx’s theory of labour, value, and exploitation under capitalism account for the production of the Mardi Gras beads?
In your own words, explain commodity fetishism and the different approaches taken to this practice by the false consciousness theory of ideology versus the cynical reason theory of ideology. Next, analyze some of the answers given by people at the Mardi Gras celebrations in New Orleans when the documentarist asks them if they know where the beads come from. Which of the responses of the Mardi Gras celebrators might be seen as an example of a false consciousness version of commodity fetishism? Why? Which seems more like a cynical reason version of commodity fetishism? Why? One way to approach this question is to choose responses which seem particularly good examples of one or the other approach to commodity fetishism and compare and contrast them. You may draw on the responses of Mardi Gras celebrators provided at any point in the documentary, but note that some excellent responses can be found in a brief sequence beginning at minute 31 of the documentary.

Sample Solution

breathing but decreased effectiveness. Pt’s chest X-ray showed cardiomegaly with mild vascular congestion and retrocardiac atelectasis.
In patients that have moderately severe COPD, their drive to breathe becomes oxygen because their body retains too much carbon dioxide. This will increase the risk of patients developing hypercapnia and requiring supplemental oxygen therapy. An increase in arterial carbon dioxide leads to an increase depth and rate of respiration. Over time, COPD patients will have high arterial levels of carbon dioxide and low levels of oxygen. The central chemoreceptors become less sensitive to these changes, consequently the stimulus to breathe is now managed by the peripheral chemoreceptors located in the carotid bodies and the aortic arch. These receptors are stimulated by low arterial levels of oxygen, and leads to hypercapnia. Thus, if supplemental oxygen is required, small changes to the rate of administration can have significant effects in those who have a hypoxic drive to breathe.
(Porth, 2011)
GERD
Gastroesophageal reflux is the backward flow of stomach contents into the esophagus without associated vomiting. It is caused by relaxation of the lower esophageal sphincter. Normal LES pressure is 10-30 mmHg and is under muscular, hormonal and neural control. In patients with GERD, the pressure is less than 10 mmHg. The decreased pressure is what allows the reflux stomach contents. Mucosal damage and erosion can result form this disease because the esophageal mucosa does not have the same protective lining mechanism as the stomach. This disease becomes chronic when the esophagus is exposed to gastric contents for a prolonged period, and an inflammatory process is initiated. The normal squamous epithelial cells are replaced with columnar epithelium, which is more resistant to damage by acidic stomach contents and supports healing of the erosions. This new epithelium is called Barrett’s

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